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If you check out the large spleen can not diagnosed with liver cirrhosis?

November 26th, 2008

My father in a hospital diagnosed with screening for five Xiao Sanyang, early cirrhosis, but in the other hospital for medical examination only when checking out the spleen is not large liver, doctors said only a large liver spleen also can be diagnosed with big . I would like to ask whether diagnosed with ???

Answer:

Patients with cirrhosis of the laboratory:
1. Blood

Decompensated more normal period of decompensation many different levels of anemia; hypersplenism, white blood cell and platelet count reduction. Secondary bacterial infection, the total number of leukocyte and neutrophil counts can be increased.

2.

In asymptomatic patients with cirrhosis and static, normal , ALT, or in some cases γ-GT increased slightly, A / G ratio slightly decreased urine urine often gall of the original increase. Activity in patients with cirrhosis often deep jaundice, inflammation of liver cell necrosis prompted more than its regeneration. Jaundice is to determine the depth of the credibility of the mark. Patients with this type of extension of the prothrombin time, the gradual lowering of plasma protein, serum aminotransferase often higher in the early, highly similar to the activities of the chronic hepatitis.

3. Electrolyte:

In asymptomatic patients with cirrhosis and static, the electrolyte can be no significant change. And the activities of decompensated cirrhosis patients, low sodium and low potassium can occur.

4. Renal:

In patients with decreased urine output, creatinine and blood urea nitrogen can be increased.

5. Blood biochemistry and immunology check:

The occurrence of hepatic encephalopathy in patients with in addition to the above-mentioned changes, can be detected increased blood ammonia, branched-chain amino acids and aromatic amino acid ratio imbalance.

6. Pathogen detection:

Hepatitis in patients with or serum can be detected in the liver biopsy based on the pathogen.

The diagnosis and treatment of
[Diagnosis]
(A) have a common cause of cirrhosis of chronic hepatitis, chronic schistosomiasis, alcoholism, malnutrition, chronic heart failure and bile pent-up Khan and metabolic disorders (such as iron, copper deposition).
(B) of the portal hypertension group of ascites, splenomegaly, hypersplenism and the formation of collateral circulation such as esophageal and gastric varices, abdominal varicose veins and hemorrhoids, such as varicose veins.
(C) of the liver function damage the performance of Nacha, fatigue, abdominal distension, bleeding tendency, pigmentation, liver palms, spider and facial telangiectasia, and so on.
(D) of the complications of upper gastrointestinal bleeding, liver coma, liver and kidney syndrome, cancer, infection, such as lung, bile duct, abdominal cavity and urinary tract infection, and so on.
[Deal]
(A) to rest, nutritious, low-fat diet, alcohol. Ammonia were higher limit of protein. There are those who Shaoyan ascites or salt-free diet. To avoid liver damage drug.
(B) of the liver drug liver music too, VitBco, VitC. Increased transaminase can give short-term Yiganling, CoA, ATP. High blood lipid lowering drugs to be as inositol, inositol lipid, and so on.
(C) hepatic coma, upper gastrointestinal bleeding, and other serious complications, the patient should be to save. There are ascites and with the exception of infection, ascites possible to lose concentration operation; have portal hypertension and hypersplenism, the conditions could be considered splenectomy and bypass surgery.

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